💀 Horrorcow Nicholas Robert Rekieta / Rekieta "Law" / Actually Criminal / @NickRekieta / "u/Early-Leopard-8351" - Polysubstance abuser, child doser, dog killer. "Lawtube pope" turned zesty Dabbleverse Redditor streamer. Swinger "whitebread ass nigga" who snuffs animals and visits 🇯🇲 BBC resorts. Legally a cuckold. Still not over his ex Aaron. Wife's bod worth $50.

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Luna's expiration date is?

  • <1 year

    Votes: 156 22.4%

  • Around 2 years

    Votes: 277 39.8%

  • 3-5 years

    Votes: 94 13.5%

  • As long as a pug lives, Karen farmer.

    Votes: 169 24.3%
  • Total voters
    696
dustnight said:
You know in light of the recent cuckening revelations by @StarTrak going all the way back to before the marriage even began, what do you think this "I will be us" was all about not long ago? 🤔
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I don’t think that changes anything. Reminder that Nick is a self-confessed porn addict since before he met Kayla, just in case anyone has a poor Nicky thing going on, he’s hardly naive. With the tattoo specially, it’s his narcissistic self-victimisation bubble where everything that happens to the family falls on him as the father. He was heavily hinting at it in his fake accountability streams after his arrest. He would throw Kayla and April under the bus, blame the entire government, then wash over it with some variant of “but hey guys, that’s on me.”
 
Balldo said:
However, I would urge people to understand that the illicit drug world is massive and ensnares all kinds of people you would never imagine in a million years would get tied to such a thing.
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Like 90% of of the offshore competitive boat racing people getting busted for running drugs (same skillset different boats) killing the entire sport. A few of them doing it to fund their sport would be expected, but enough of them that getting caught would kill the sport? That's an unexpected scale.
 
Terror_Mouse said:
I think he might legitimately have narcolepsy, but I feel like you don't treat it with a whole bunch of alcohol and stubbornly refusing to try and sleep normal hours. I think he probably has it, but uses it as an excuse to enable behavior instead of changing his lifestyle to try and minimize the disruption it has on his life.
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Interesting finds here ....

Abstract: Narcolepsy induced by chronic heavy alcohol consumption: a case report​

Summary​

Narcolepsy is a chronic neurological disorder, characterized by uncontrollable excessive daytime sleepiness, cataplectic episodes, sleep paralysis, hypnagogic hallucinations, and night time sleep disruption. The paper reviewed the related literature and reported a case of long-term drinking induced narcolepsy which was significantly improved after treatment with paroxetine and dexzopiclone.

1.1. Preadmission history​

The patient was a 39-year-old married male of Han ethnicity who worked as a daytime truck driver. In 2005, because of work-related stress he began to drink heavily in the evenings, consuming 250 to 500 ml of spirits. He was often intoxicated and typically slept only 3 to 4 hours before waking to start work the following day. He subsequently developed acute daytime drowsiness for periods of 10 minutes at a time while walking or driving. He also started experiencing episodes of sudden loss of motor power while fully conscious (cataplexy) that lasted for a few seconds at a time. And he had nighttime insomnia and nightmares. The symptoms were initially relatively mild, but he did have a car accident during an episode of narcolepsy. Over the next 3 years the symptoms gradually got worse; the intensity and frequency of episodes appeared related to the amount of alcohol he consumed. After 3 years of these symptoms he changed to a less stressful driving job and was able to gradually decrease his drinking without serious withdrawal symptoms. Over the subsequent 2 years the frequency of acute daytime drowsiness and cataplectic episodes decreased somewhat, but his nighttime insomnia remained. A two-week admission to a neurology inpatient service over this period did not result in any improvement in his condition.

1.2. Psychiatric admission and follow-up​

In October 2011 he was voluntarily admitted to our hospital. He reported a 10-year history of alcohol abuse that had fluctuated in severity over time but he had completely stopped alcohol consumption 2 years previously because he felt drinking exacerbated his narcolepsy and cataplexy. Other than expressing worry about his narcolepsy and cataplexy there were no positive findings on the routine mental status examination on admission. He had elevated blood lipids but routine blood chemistry, CT, MRI, EEG and EKG examinations found no other abnormalities. A full sleep study found a total sleep time of 451 minutes, sleep efficiency of 83% (without sleep apnea), and a REM latency of 10.5 minutes; the percentage of REM was 69.8%, the percent lucid intervals was 17.5%, and the percentages of S1, S2 and S3 sleep were 3.8%, 0.5% and 8.4%, respectively.

Given an admission diagnosis of narcolepsy, the patient was treated with 20 mg paroxetine each morning and 3 mg dexzopiclone each evening. After one week of treatment, the number of both narcoleptic episodes and cataplectic episodes decreased. After one month of treatment the symptoms had resolved completely so he was discharged. He remained off alcohol after discharge. He continued the paroxetine and dexzopiclone for two months after discharge and then gradually stopped the medications. However, one month later the narcoleptic and cataplectic symptoms returned so he restarted the paroxetine (but not the dexzopiclone) and continued to take it daily. His narcoleptic and cataplectic symptoms resolved, his nocturnal sleep normalized (7 to 8 hours per night without nightmares), and he was able to return to fulltime work. The improvement has continued to the present – 6 months after restarting the paroxetine.

Narcolepsy is a rare, chronic nervous system disease that usually includes acute daytime drowsiness, episodic cataplexy, sleep paralysis, hypnagogic hallucination and a disrupted sleep cycle. In a report of 35 patients with narcolepsy, Bai and colleagues[1] found that most patients had cataplexy, sleep paralysis and hypnagogic hallucinations and that the duration of the paroxysmal daytime sleep, which varied considerably, was longer in those with an earlier age of onset. Several abnormalities on the polysomnography examination (i.e., sleep EEG study) are indicative of narcolepsy:[2] a) sleep latency shorter than 10 minutes; b) REM latency shorter than 20 minutes; c) multiple sleep latency test (MSLT) showing an average sleep latency shorter than 5 minutes; or d) two or more episodes of sleep beginning with REM.

Narcolepsy was first reported and named by Gelineau in 1880 but the etiology remains unknown. It can be hereditary (in 10 to 30% of cases) or occur following various results to the central nervous system including cerebral trauma, viral infection, immune dysfunction, brain tumors, multiple sclerosis, or severe emotional stress. Chronic heavy alcohol use can cause widespread damage to the central nervous system so it may also be a cause of narcolepsy. The exact mechanism for inducing paroxysmal sleep is uncertain, but studies have shown that stimulation of the posterior hypothalamus inhibits the reticular activating system and, thus, induces sleep.[2] The finding of early initiation of REM suggests that melatonin may play a role in the disorder.[3] Some patients have elevated dopamine D2 receptor and L-dopa (the metabolite of dopamine) and increased serotonin activity in the amygdaloid nucleus or the corpus striatum. Many individuals with narcolepsy have a specific HLA-2 allele (human leukocyte antigen DQB1*0602 allele).[4]

The patient described in this case report had several of the cardinal symptoms of narcolepsy: a) uncontrollable excessive daytime drowsiness; b) cataplexy (which was more serious with emotional excitation); c) disrupted nocturnal sleep; and d) REM latency of less than 20 minutes. These clinical characteristics are similar to those described in the literature and are listed as diagnostic criteria of narcolepsy in the 1997 International Classification of Sleep Disorders.[5] Despite a long history of drinking, the patient had stopped drinking two years prior to admission, so at the time of admission he did not have a concurrent diagnosis of alcohol abuse, alcohol dependence or alcohol withdrawal.[6]

The treatment of narcolepsy is mainly symptomatic, aimed at suppressing symptoms and improving patients' quality of life.[7] The most common modalities include stimulants and behavioral therapy to reduce acute daytime drowsiness, antidepressants to decrease episodes of cataplexy and other REM-related symptoms, and sedatives to improve the quality of nocturnal sleep.[8]–[10] In this case the combination of the antidepressant paroxetine (a selective serotonin reuptake inhibitor) and dexzopiclone (a non-benzodiazapine hypnotic) controlled the symptoms but – despite full withdrawal from alcohol – the narcolepsy and catalepsy symptoms recurred when the medications were stopped so the patient had to be continuously treated with paroxetine. This suggests that chronic heavy alcohol use precipitated the initial episodes of narcolepsy and catalepsy – perhaps by inhibiting the central nervous system's reticular activating system – but was no longer necessary to sustain the disorder after it had started. Alternatively, it is also possible that the alcohol abuse and narcolepsy were initially independent comorbid conditions.

TLDR: A medical abstract studied by a doctor who followed the case of a 39 year-old male and how his heavy drinking effected his narcolepsy. Alcohol didn't help .... only made it worse!

The Effects of Narcolepsy and Alcohol​

As with any substance use, alcohol may increase the undesirable symptoms of narcolepsy. Though it may be used to self-medicate for a better night’s sleep, alcohol can obstruct a good night’s rest.​

Article at a Glance:​

  • Alcohol use is not proven to be a distinct cause of the sleep disorder narcolepsy.
  • However, the use of alcohol could create narcolepsy-like symptoms for the person drinking alcohol.
  • If a person with narcolepsy ingests alcohol, they may increase the challenging symptoms they are already dealing with.

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Can Alcohol Consumption Cause Narcolepsy?​

Alcohol is commonly thought of as a sedating or calming substance. While alcohol may induce a sleepy sensation in people who have consumed a few drinks, sleep quality tends to decrease after ingesting alcohol. Someone who drinks a glass or two of wine may feel drowsy after drinking. However, this is not likely, as alcohol consumption makes a person more likely to wake up during the night, worsening their overall quality of sleep.

This sleep disruption typically occurs during the second half of the sleep period once alcohol’s relaxing effect wears off. The disruption keeps the body from experiencing the critical REM state and may lead to daytime fatigue and sleepiness. A continued cycle of consumption and REM disturbances can lead to symptoms similar to those experienced with narcolepsy, like:
  • Daytime sleepiness
  • Disrupted REM sleep
  • Vivid dreams
  • Possible hallucinations caused by lack of sleep
Alcohol should be avoided before or close to bedtime. It prevents people from getting the deep sleep and REM sleep they need because alcohol keeps them in the lighter stages of sleep.

Hypocretin is an important chemical produced by the brain for regulating wakefulness and REM. Lack of the chemical in the brain can cause narcolepsy-like symptoms.

Alcohol-Induced Narcolepsy​

Alcohol-induced narcolepsy is caused by alcohol consumption and characterized by overwhelming daytime drowsiness and sudden attacks of sleep. If the urge becomes irresistible, patients fall asleep for periods lasting from a few minutes to several hours. These episodes of random sleep can occur at inappropriate times and places such as while driving, at bars, work and restaurants. In turn, people with sleep disorders are also at risk to develop substance use disorders in an attempt to induce sleep. Most often, thesubstances of abuse for individuals with sleep disorders are alcohol, narcotic medications and benzodiazepines.

Alcohol-induced narcolepsy should not be confused with passing out from excessive drinking, which usually consists of a person falling asleep, waking up, drinking more, actively engaging in activities and then falling asleep again. This cycle may repeat throughout the day and into the evening.

Co-Occurring Treatment of Narcolepsy With Alcohol Abuse​

Patients who meet the formal diagnostic criteria for narcolepsy and either alcohol use disorderor addiction would need to treat both disorders simultaneously. Trying to address one disorder while ignoring the other would be counterproductive and result in negative effects.

Someone with these co-occurring diagnoses requires combined treatment. The purpose of the combined treatment is a long-term success, including aftercare planning. Ultimately, with a comprehensive treatment approach, people who suffer from narcolepsy and alcohol abuse can embrace a healthy life in recovery.

Alcohol and Narcolepsy​

Alcohol and narcolepsy have an impact on sleep. Narcolepsy is a sleep disorder that affects the sleep-wake cycle, and the consumption of alcohol can produce sleep disturbances and symptoms similar to those of narcolepsy.

Consuming alcohol initially creates feelings of relaxation, so it is sometimes used as a sleep aid by people with sleeping problems. Although alcohol and narcolepsy can create similar sleep-related symptoms, there are key differences in the ways they work.

​

About Alcohol Use​

Moderate alcohol use is usually defined as one drink per day for women and two drinks per day for men. The abuse of alcohol is underlined by an unhealthy consumption of alcohol that is driven by both genetic and environmental factors, which sometimes results in alcohol addiction.

Alcohol is a substance that, when used moderately or heavily, can cause different sleep disturbances and problems. Even light alcohol use may affect a person’s sleep quality and contribute to sleep-related difficulties, including symptoms of conditions like narcolepsy or insomnia. People react differently to alcohol consumption and experience varying levels of mental and emotional effects.

​

What’s the Connection Between Alcohol Use and Narcolepsy?​

Someone who experiences narcolepsy and trouble sleeping at night may use alcohol as a sleep aid. However, alcohol use and narcolepsy both negatively affect sleep. Alcohol consumption may contribute to extreme fatigue and a condition called alcohol-induced narcolepsy, although it is unlikely to cause narcolepsy itself. Rather, alcohol use and narcolepsy both can inhibit deep sleep and produce similar daytime symptoms resulting from poor-quality sleep at night.

How Alcohol Use and Narcolepsy Affect Sleep​


Alcohol use produces feelings of relaxation and sleepiness and acts as a sedative on the central nervous system. However, drinking alcohol in excess or right before bed may cause sleep disturbances.

Alcohol use affects sleep by making it more difficult to enter the rapid eye movement (REM) stage of sleep. Alcohol interferes with the neurotransmitters (chemical signals) in the brain that are responsible for regulating sleep and wake cycles.

Because alcohol at first causes sleepiness, it may cause a person to enter the stage of deep sleep earlier than normal, but it will create an imbalance in the sleep stages throughout the night. Alcohol-related disruptions to the normal sleep cycle may make it difficult to feel well-rested. A person may also experience other symptoms, including some similar to narcolepsy:
  • Daytime sleepiness or drowsiness
  • Vivid dreams
  • Possible hallucinations from lack of sleep
Narcolepsy affects sleep in a way similar to alcohol use, as it also interferes with the normal cycles of sleep. A shortage of hypocretin may cause symptoms such as excessive daytime sleepiness. Other symptoms of narcolepsy include:
  • Cataplexy — A sudden loss of muscle tone and function
  • Sleep paralysis — The inability to move or speak between episodes of sleep and wakefulness
  • Hallucinations — Visual or auditory perceptions of things that are not truly there, right before falling asleep or right after waking up (sometimes called hypnagogic hallucinations)
  • Automatic behaviors — Including falling asleep for several seconds during the day while performing tasks like eating or driving, and having no memory of it happening

Alcohol as a Sleep Aid​

Alcohol use may also lead to insomnia, a sleep disorder characterized by the inability to fall asleep at night. Many studies have shown an association between alcoholism and insomnia. A person may self-medicate their insomnia by drinking more alcohol, and end up in a complicated cycle between alcohol use and insomnia.

Similarly, people with narcolepsy may experience a problematic cycle by using alcohol to counteract the inability to fall asleep at night. People on MyNarcolepsyTeam have written about the interaction between their narcolepsy and alcohol use. “It’s hard using alcohol to cope. You get a little bit of a pickup at first ... and then you crash hard going into the next day,” wrote one member.

TLDR: Narcolepsy combined with the abuse of alcohol can lead to a wide range of even more health problems including hallucinations, loss of muscle tone and function, visual and auditory hallucinations, and insomnia.

EDIT TO ADD:

What I imagine going on a date with Nick was like ....

 
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I would not be surprised if Nick is diagnosed with some kind of Korsakoff Syndrome adjacent degenerative mental impairment. The drugs will impact the kids when papa becomes a drooling vegetable.
 
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thebigdream said:
In a functioning legal system, it's obvious Nick doesn't have a case here - Aaron can't lose all right to legal protections just because he himself engaged in misconduct with a third party, that would be absurdly unfair. But given how the courts have licked his boots before, I don't blame Nick for taking a shot.
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Aaron has a CIVIL RIGHTS violation lawsuit.
Much Like that Maya case being rerversed=I mean, come on, holding a hospital liable for a NON-Patient suicide? Where would hospital liability ever stop if such precedent were allowed...
 
ohhhhhhhhhhhhhhh

Nick cut his hair short and has been gelling it because that's how he wore his hair when he still had youth

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this is beyond pathetic
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He got another haircut like today? Nick - your hair is waaaaaaay too thin to have it that short. You're all scalp, buddy

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Terror_Mouse said:
The fact he skated by on what the charges should have been--and the ones that never even came up!--and got Kayla/April off the hook, and has essentially been completely ignored when it comes to his "don't do drugs/break the law" conditions... yeah, he won on the legal front.
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Absolutely no lawyer who looked at this case, from Barnes to Sean, anticipated he'd get anything more than a deferred adjudication with maybe a year of probation. He got a near the top of the guidelines sentence.
 
daffodils said:
Elissa has struck gold
https://youtube.com/watch?v=nWijnFD4I9I--
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The way he speaks for his audience (who conveniently agree with him) is hilariously similar to DSP.

Personally, I wish this guy would have his probation dropped so he can go back to doing all the drugs he wants. It's funnier that way. Well, they should remove the kids from the situation first.
 
AnOminous said:
Absolutely no lawyer who looked at this case, from Barnes to Sean, anticipated he'd get anything more than a deferred adjudication with maybe a year of probation. He got a near the top of the guidelines sentence.
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@Terror_Mouse kinda missed the sarcasm in my post he replied to.

Anyone who followed the case knew this was the first offender pack every dumbfuck Nigger in Minnesota gets these days.
Nick won nothing, on the contrary. He lost every single fight he picked for the last 16 months.
 
Captain Manning said:
That's great dude. I know all that. The CA still let him slide on two counts of child endangerment,
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No they didn't. They said they would drop those charges if he plead guilty to a felony, saved a trial and went on probation for 5 years. The plea deal was predictable. The endangerment stems from guns and drugs which are gone as part of probation. No drugs, no drinking, no guns achieves the objective of an endangerment conviction. Even if he plead guilty to those 2 misdemeanor charges, he would have the same punishment. It was never going to be worse than what he got. His children will largely be adults by the time he can drink again.
 
Gobermental Supervisor said:
Nick won nothing, on the contrary. He lost every single fight he picked for the last 16 months.
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He spent tens of thousands of dollars and over a year fighting for the worst result he could get unless the prosecutor did something nobody expected and didn't just use the endangerment charge to coerce a plea to the drug felony.

If he'd just gone for what the likely offer was going to be, something like a year's probation with adjudication deferred, he'd already be done with it for maybe a couple grand to some flat fee lawyer. Then they'd have dismissed everything outright. Instead he has a felony conviction and five years probation, although he's lucked out with a useless probation officer.
 
That was an interesting veiled comment by Rekieta in context of his HRO defence, saying anyone litigating against you should be humiliated.
Around 1:23:00.
 
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